A-) H\u0130POF\u0130Z: Beyinde, temel kemikteki T\u00fcrk e\u011feri denilen \u00e7ukur i\u00e7inde bulunur. \u0130nce bir sap ile hipotolamusa ba\u011fl\u0131d\u0131r. Di\u011fer i\u00e7 salg\u0131 bezlerinin \u00e7al\u0131\u015fmas\u0131 \u00fczerinde \u00f6nemli d\u00fczenleyici g\u00f6revi vard\u0131r. \u00d6n, ara ve arka lob olmak \u00fczere 3 k\u0131s\u0131mdan olu\u015fmu\u015ftur. Ara lob insanda sadece fet\u00fcste g\u00f6r\u00fcl\u00fcr. Ergin fertte iz halinde kal\u0131r. \u00d6nve arka loblar epitel, ara lob sinir dokudan yap\u0131lm\u0131\u015ft\u0131r.<\/p>\n
1) \u00d6n Lob Hastal\u0131klar\u0131
\n \u00d6n lob, b\u00fcy\u00fcme hormonu (somoatotropik) salg\u0131layarak kemiklerin ve genel olarak v\u00fccudun b\u00fcy\u00fcmesini kontrol eder.<\/p>\n
a) AKROMEGAL\u0130 (ORANTISIZ B\u00dcY\u00dcME)
\n Hipofizin \u00f6n lobundaki b\u00fcy\u00fcmehormonu salg\u0131layan asidofil h\u00fccrelerinin a\u015f\u0131r\u0131 art\u0131\u015f\u0131 ayn\u0131 hastal\u0131\u011f\u0131n 2 g\u00f6r\u00fcn\u00fcm\u00fcn\u00fc ortaya \u00e7\u0131kar\u0131r. B\u00fcy\u00fcmesi sona ermi\u015f olan yeti\u015fkin ki\u015fide akromegali; organizmas\u0131 geli\u015fmekte olan ki\u015fide devlik. Bu 2 g\u00f6r\u00fcn\u00fcm, \u00e7e\u015fitli bi\u00e7imlerde biraraya gelebilirler.
\n Akromegali terimi, Yunanca “alero” (u\u00e7) ve “mega” (b\u00fcy\u00fck) s\u00f6zlerinden t\u00fcretilmi\u015ftir ve hastal\u0131\u011f\u0131n tipik bi\u00e7iminde g\u00f6r\u00fclen bi\u00e7im bozukluklar\u0131n\u0131 \u00e7ok iyi anlat\u0131r. Hastal\u0131k, ba\u015fl\u0131ca 3 grup belirti verir. \u00d6nce, \u00f6zellikle \u00fcyeleri etkileyen ve hacim artmas\u0131 bi\u00e7iminde kendini g\u00f6steren bi\u00e7im bozukluklar\u0131; sonra bir hipofiz urunu d\u00fc\u015f\u00fcnd\u00fcrebilecek sinirsel belirtiler; en son olarak da klinik belirtilerle yans\u0131yabilen i\u00e7 salg\u0131 ve metabolizma bozukluklar\u0131d\u0131r.
\n Tipik bi\u00e7iminde akromegali, yeti\u015fkinlerde yani b\u00fcy\u00fcme sona erdikten sonra g\u00f6r\u00fcl\u00fcr. Gen\u00e7 erkeklerde ve kad\u0131nlarda daha s\u0131k rastlan\u0131r.
\n \u00d6n hipofizin asidofil h\u00fccrelerinden kaynaklanan bir adenomdur. Adenom:Bir bezden kaynaklanan ve yap\u0131s\u0131 bu bezi and\u0131ran bir urdur.S\u00f6z konusu ur iyicildir ama yerle\u015fme yeri ciddi sonu\u00e7lara yol a\u00e7abilir ve b\u00f6ylece * yeri bak\u0131m\u0131ndan k\u00f6t\u00fcc\u00fcl * duruma gelebilir.
\n Bu asidofil adenom, etkisi akromegalideki bi\u00e7im bozukluklar\u0131n\u0131 yaratan, a\u015f\u0131r\u0131 b\u00fcy\u00fcme hormonu (STH) salg\u0131lar.
\n Akromegalili bir hastada el ve ayaklar\u0131n b\u00fcy\u00fcmesi
\n Akromegalili bir hastan\u0131n kafa filminin \u00e7izimi.G\u00f6r\u00fclenler: 1.Al\u0131n kemi\u011fi sin\u00fcs\u00fcn\u00fcn hacmi artm\u0131\u015f – 2.T\u00fcrk eyeri geni\u015flemi\u015f – 3.Kafa kubbesi kal\u0131nla\u015fm\u0131\u015f – 4. Artkafan\u0131n d\u0131\u015fa \u00e7\u0131k\u0131nt\u0131s\u0131 a\u015f\u0131r\u0131la\u015fm\u0131\u015f – 5.Alt \u00e7ene \u00f6ne \u00e7\u0131km\u0131\u015f.<\/p>\n
b) DEVL\u0130K (J\u0130GANT\u0130ZM)
\n Ya\u015f\u0131na g\u00f6re boyu, kendi ya\u015f\u0131dakilerin boy ortalamas\u0131n\u0131n en az 3 ya\u015f b\u00fcy\u00fc\u011f\u00fcn\u00fcn boyu kadar olan ki\u015fiye “dev” denir. Devlik (jigantizm) ender rastlanan bir hastal\u0131kt\u0131r. Anormal b\u00fcyme, genel olarak ergenlikle birlikte ba\u015flar. Devlikte hastal\u0131k, hipofizin \u00f6n b\u00f6l\u00fcm\u00fcndeki a\u015f\u0131r\u0131 STH (somoatotropik) salg\u0131lamas\u0131 sonucu olu\u015fur. Ayr\u0131ca \u00f6n hipofizin asidofil h\u00fccrelerinden kaynaklanan adenom mutlak bulunmayabilir; \u00e7e\u015fitli sinirsel bozukluklar devli\u011fe yola\u00e7abilir. <\/p>\n
c) CUSH\u0130NG HASTALI\u011eI
\n Cushing hastal\u0131\u011f\u0131, kortikotrop hormonunun (ACTH) a\u015f\u0131r\u0131 salg\u0131lanmas\u0131 sonucu ortaya \u00e7\u0131kar. Bu hastal\u0131ktan s\u00f6z etmeden \u00f6nce \u00fcst\u00fcnde durulmas\u0131 gereken bir nokta vard\u0131r. Genelde bu hastal\u0131k, \u00f6zellikle b\u00f6brek \u00fcst\u00fc kabu\u011fu bezlerinin a\u015f\u0131r\u0131 \u00e7al\u0131\u015fmas\u0131n\u0131 g\u00f6steren belirtilerle (hiperkotisim) yani glukokortikoit hormonlar\u0131n\u0131n a\u015f\u0131r\u0131 salg\u0131lanmas\u0131yla yans\u0131r. B\u00f6brek \u00fcst\u00fc kabu\u011fu bezlerinin a\u015f\u0131r\u0131 \u00e7al\u0131\u015fmas\u0131n\u0131n k\u00f6keninde klinikte Cushing sendromu diye adland\u0131r\u0131lan ortak bir tabloya yol a\u00e7an, bir\u00e7ok neden bulunabilir.
\n Cushing hastal\u0131\u011f\u0131, nedeni hipotolamus-hipofiz d\u00fczeyinde yerle\u015fen bir cushing sendromudur. Bir hipofiz uru yada ur olmaks\u0131z\u0131n hipotolamus d\u00fczensizli\u011fi s\u00f6z konusu olabilir.
\n Cushing hastal\u0131\u011f\u0131na yol a\u00e7an hipofiz uru ya k\u00f6t\u00fcc\u00fcld\u00fcr (\u00e7ok ender olarak) yada kromofob yadsa bazofil h\u00fccrelerden geli\u015fen bir adenomdur.
\n Cushing sendromu tipinde bir b\u00f6brek \u00fcst\u00fc bezleri kabu\u011funun a\u015f\u0131r\u0131 geli\u015fmesiyle birlikte olan hipofiz urunun \u00f6zel bir bi\u00e7imi, bu urun bir b\u00f6brek \u00fcst\u00fc bezinin \u00e7\u0131kar\u0131lmas\u0131ndan (tam yada tam olmayan) sonra g\u00f6r\u00fclmesidir. Derideki pigment art\u0131\u015f\u0131, bu bi\u00e7imde \u00e7ok iyi bir belirtidir. Ur belirtilerinin bir \u00f6zelli\u011fi yoktur. Buna kar\u015f\u0131l\u0131k ACTH’\u0131n plazma d\u00fczeyinin \u00e7ok y\u00fcksek olmas\u0131 ve lizin-vazopressin uyar\u0131s\u0131na yan\u0131t\u0131n son derece belirgin olmas\u0131, \u00e7ok iyi belirtilerdir.<\/p>\n
d) TAM VE KISM\u0130 \u00d6N H\u0130POF\u0130Z YETMEZL\u0130\u011e\u0130
\n Hipofizin \u00f6n lobunun yetmezli\u011fi (hipopit\u00fcitarizm), h\u00fccrelerin tamam\u0131n\u0131n (yada tama yak\u0131n\u0131n) yada birb\u00f6l\u00fcm\u00fcn\u00fcn etkilenmesine g\u00f6re, de\u011fi\u015fik \u015fekillerdedir. H\u00fccrelerin tamam\u0131 veya tama yak\u0131n\u0131 etkilenmi\u015fse hastal\u0131k tam \u00f6n hipofiz yetmezli\u011fi (panhipopit\u00fcitarizm) diye adland\u0131r\u0131l\u0131r. 2. durumda, ksmi \u00f6n hipofiz yetmezli\u011fi s\u00f6z konusudur. Bunlar de\u011fi\u015fik g\u00f6r\u00fcn\u00fcmler verebilirler oysa tam \u00f6n hipofiz yetmezli\u011finin olu\u015ftu\u011fu \u015fekil olduk\u00e7a kesindir. Hipofiz \u00f6n lobunun b\u00fct\u00fcn\u00fcyle yetmezli\u011fi olan bu hastal\u0131k, \u00f6n hipofize ba\u011f\u0131ml\u0131 \u00e7evresel i\u00e7 salg\u0131 bezlerinin ikincil eksikli\u011fiyle (yetmezli\u011fi) yans\u0131r: Troid bezi, b\u00f6brek \u00fcst\u00fc kabu\u011fu bezleri, cinsellik bezleri.
\n Hastal\u0131\u011f\u0131n nedeni \u00e7e\u015fitlidir, ama ba\u015fl\u0131ca naden \u00f6n hipofizin kanlanma eksikli\u011fiyle olu\u015fan doku \u00f6l\u00fcm\u00fc ve hipofiz urlar\u0131d\u0131r. En tipik \u00f6n hipofiz yetmezli\u011fi, \u00e7ok kanl\u0131 bir do\u011fumdan sonra, atardamar bas\u0131nc\u0131n\u0131n \u00f6nemlid\u00fc\u015f\u00fc\u015f\u00fcyle birlikte hipofiz \u00f6n lobunun kanlanma azl\u0131\u011f\u0131ndan \u00f6t\u00fcr\u00fc doku \u00f6l\u00fcm\u00fcne u\u011framas\u0131 sonucu oland\u0131r. Ayr\u0131ca bu etmenin, ba\u015flang\u0131\u00e7ta hastal\u0131\u011f\u0131n ortaya \u00e7\u0131k\u0131\u015f\u0131n\u0131 kolayla\u015ft\u0131r\u0131c\u0131 bir etmen oldu\u011fu san\u0131lmaktad\u0131r; \u00e7\u00fcnk\u00fc \u00f6n hipofiz yetmezli\u011finin kar\u015f\u0131lanamaz bir duruma gelmasi ancak bir\u00e7ok gebelikten sonra ortaya \u00e7\u0131kar.
\n Hipofiz doku \u00f6l\u00fcm\u00fcne u\u011framas\u0131na yol a\u00e7an \u00f6teki nedenlere \u00e7ok ender rastlan\u0131r.
\n Hipofizle ilgili yada hipotolamus-hipofiz b\u00f6lgesiyle ilgili bir ur (k\u00f6t\u00fcc\u00fcl ur yada adenom) dahi tam \u00f6n hipofiz yetmezli\u011fine yada k\u0131smi \u00f6n hipofiz yetmezli\u011fine yol a\u00e7abilir. <\/p>\n
2) Arka Lob Hastal\u0131klar\u0131<\/p>\n
a) Arka Hipofiz Yetmezli\u011fi: \u015eEKERS\u0130Z \u015eEKER HASTALI\u011eI
\n \u015eeker hastal\u0131\u011f\u0131n\u0131n tersine, \u015fekersiz \u015feker hastal\u0131\u011f\u0131nda, idrar \u015fekerli de\u011fildir. Hastal\u0131\u011f\u0131n ba\u015fl\u0131ca belirtileri 24 saatlik idrar miktar\u0131n\u0131n a\u015f\u0131r\u0131 art\u0131\u015f\u0131 (poli\u00fcri) ve hastay\u0131 \u00f6nemli miktarlarda s\u0131v\u0131 i\u00e7meye zorlayan a\u015f\u0131r\u0131 susuzluktur (polidipsi).
\n Antidi\u00fcretik hormon eksikli\u011fin hipotolamus- hipofiz sistemi d\u00fczeyinde yani ya hipotolamusun g\u00f6rme sinirleri diski \u00fcst\u00fc ve kar\u0131nc\u0131klar yan\u0131 \u00e7ekirdekleri d\u00fczeyinde ya hipofizin arka b\u00f6lgesinde yada bunlar\u0131 birle\u015ftiren olu\u015fumlarda yerle\u015fen bozunlar sonucu ortaya \u00e7\u0131kabilir.
\n Antidi\u00fcretik hormon eksikli\u011fi ile olu\u015fan \u015fekersiz \u015feker hastal\u0131\u011f\u0131n\u0131n nedenleri pek \u00e7oktur. Genel olarak s\u00f6ylersek merkezi sinir sisteminde g\u00f6zlenen b\u00fct\u00fcn bozunlar, \u015fekersiz \u015feker hastal\u0131\u011f\u0131na yol a\u00e7abilirler.
\n Antidi\u00fcretik hormonun fizyolojik rol\u00fcn\u00fc an\u0131msarsak, hastal\u0131\u011f\u0131n mekanizmas\u0131n\u0131 anlamak \u00e7ok kolayd\u0131r. Bu hormon olmazsa, b\u00f6brek suyututamaz, s\u00fczemez ve su serbest\u00e7e ka\u00e7ar; bundan dolay\u0131 da idrar miktar\u0131 \u00e7ok artar. Bunun sonucunda, normal olarak idrar yoluyla at\u0131lan maddelerde bir seyrelme olur. Suyun b\u00f6brekler yoluyla organizmadan ka\u00e7\u0131\u015f\u0131, kar\u015f\u0131t mekanizman\u0131n olu\u015fumuna neden olur ve hastay\u0131 i\u00e7meye iten bir susuzluk duygusu ba\u015flar.
\n Baz\u0131 genel hastal\u0131klar merkezi sinir sisteminde \u00f6zellikle hipotolamus-hipofiz b\u00f6lgesinde, antidi\u00fcretik hormonun salg\u0131lanmas\u0131n\u0131 azaltan bozunlara yol a\u00e7abilirler. Bu konuda Besnier-Boeck-Schaumann hastal\u0131\u011f\u0131 yada sorkayidoz ve histiyosit hastal\u0131klar\u0131 say\u0131labilir. \u00d6zellikle merkezi sinir sistemi hastal\u0131klar\u0131 da \u015fekersiz \u015fekere neden olabilir.
\n \u00c7e\u015fitli t\u0131p ve yerle\u015fimdeki urlar \u015funlard\u0131r;
\n -hipofiz urlar\u0131 -kranyofarenjiyomalar -epifiz urlar\u0131<\/p>\n
B) TRO\u0130D:G\u0131rtla\u011f\u0131n \u00f6n k\u0131sm\u0131na yerle\u015fmi\u015f kalkan bi\u00e7iminde, bir bez olup kan damarlar\u0131yla beslenen 2 lobdan olu\u015fur. Herbir lobda falikul denilen kesecikler ve i\u00e7lerinde kollodial madde bulunur.Troid bezi 2 hormon salg\u0131lar: Tiroksin, kalsitonin.<\/p>\n
a)GUATR
\n Tiroksin hormonunun bile\u015fiminde tirozin aminoasiti ile birlikte iyot bulunur. \u0130yot yetersizli\u011finde sentezlenemez ve bu durumda troid bezi normalden \u00e7ok \u00e7al\u0131\u015farak \u015fi\u015fer. Guatr denilen hastal\u0131k olu\u015fur. Guatr \u00e7evresel etkenlerden kaynaklanan bir hastal\u0131k olup al\u0131nan besinlerde iyot halinde ortaya \u00e7\u0131kar. Suya potasyum iyod\u00fcr kat\u0131larak g\u00f6r\u00fclme s\u0131kl\u0131\u011f\u0131 azalt\u0131labilir.
\n Tiroksin hormonu h\u00fccre metabolizmas\u0131n\u0131, yani t\u00fcm v\u00fccut h\u00fccrelerindeki metabolik olaylar\u0131n h\u0131z\u0131n\u0131 d\u00fczenler. Me tabolizma faaliyetlerinde kullan\u0131lacak oksijen miktar\u0131n\u0131 ayarlar.<\/p>\n
Tiroksinin Fazla Oranda Salg\u0131lanmas\u0131 Halinde;<\/p>\n
* H\u00fccrelerde solunum h\u0131zlan\u0131r, h\u00fccreler daha \u00e7ok oksijen kullan\u0131r.
\n * Bazal metabolizma h\u0131z\u0131 artar.
\n * Dola\u015f\u0131m h\u0131zlan\u0131r, kan bas\u0131nc\u0131 artar,kalp at\u0131\u015flar\u0131 h\u0131zlan\u0131r.
\n * Fazla art\u0131k madde \u00fcretir ve v\u00fccut \u0131s\u0131s\u0131 y\u00fckselir. S\u00fcrekli terleme g\u00f6r\u00fcl\u00fcr.
\n * D\u0131\u015f guatr g\u00f6r\u00fcl\u00fcr. Belirtileri: \u00e7ok terleme kendilerini a\u015f\u0131r\u0131 derece s\u0131cak hissederler, ald\u0131klar\u0131 besini \u00e7ok \u00e7abuk kulland\u0131klar\u0131ndan kilo kaybederler, ayr\u0131ca y\u00fcksek tansiyon, sinir gerginli\u011fi, a\u015f\u0131r\u0131 hassasl\u0131k, kas zay\u0131fl\u0131\u011f\u0131, titreme, g\u00f6z\u00fcn d\u0131\u015far\u0131 do\u011fru f\u0131rlamas\u0131 (ekzoftalmi) <\/p>\n
Tiroksinin Az Salg\u0131lanmas\u0131 Halinde;
\n \u00c7ocuklarda KRET\u0130N\u0130ZM, erginlerde M\u0130KSODEMA denilen hastal\u0131klar g\u00f6r\u00fcl\u00fcr. <\/p>\n
a) KRET\u0130N\u0130ZM
\n \u00c7ocuklarda dil geni\u015f, al\u0131n buru\u015fuk, kemikler \u00e7arp\u0131kt\u0131r.
\n Belirtileri; zeka gerili\u011fi, c\u00fccelik, e\u015feysel organlar\u0131n geli\u015fmemesi<\/p>\n
a) M\u0130KSODEMA
\n Erginlerde tiroksin azl\u0131\u011f\u0131nda ortaya \u00e7\u0131kan miksodema denilen hormonal bozuklu\u011fun ba\u015fl\u0131ca belirtileri;<\/p>\n
a) Metabolik faaliyetlerde yava\u015flama, bazal metabolizmada azalma.
\n b) Nab\u0131z yava\u015flar, fiziksel enerji kayb\u0131 yani uyu\u015fukluk g\u00f6r\u00fcl\u00fcr.
\n c) V\u00fccut \u0131s\u0131s\u0131 d\u00fc\u015fer, hasta devaml\u0131 \u00fc\u015f\u00fcr.
\n d) \u0130\u015ftah normaldir fakat besinleri normal kullanamad\u0131\u011f\u0131ndan \u015fi\u015fmanlama e\u011filimindedir.
\n e) Kaslarda zay\u0131fl\u0131k, el, ayak, hatta t\u00fcm v\u00fccutta \u015fi\u015fkinlikler g\u00f6r\u00fcl\u00fcr, deri alt\u0131nda su ve Na+ toplan\u0131r.
\n f) Zihinsel gerilemeler, k\u0131l d\u00f6k\u00fclmeleri g\u00f6r\u00fcl\u00fcr. Sa\u00e7 uzamas\u0131 duraklar.
\n g) Kanda kolesterol artar, dokular aras\u0131 s\u0131v\u0131da lenfte su ve sodyum miktar\u0131 y\u00fckselir. V\u00fccut \u015fi\u015fer.<\/p>\n
C) PARATRO\u0130D: Parathormon yada paratirin denilen bir hormon yapar. Bu hormon kan ve kemiklerdeki Ca ve P metabolizmas\u0131n\u0131 d\u00fczenler.\u0130\u00e7 dengenin kurulmas\u0131na yard\u0131mc\u0131 olur. Ca kemik te\u015fekk\u00fcl\u00fcnde, kas ve sinir sisteminin normal \u00e7al\u0131\u015fmas\u0131nda kan\u0131n p\u0131ht\u0131la\u015fmas\u0131nda, aktif ta\u015f\u0131mada t\u00fckr\u00fck bile\u015fiminde gerekli olan bir maddedir.
\n Parathormonun Az Salg\u0131lanmas\u0131 Halinde;
\n Kandaki Ca miktar\u0131 azal\u0131r. Kemiklerde ise artar. Kanda Ca azalmas\u0131 halinde TETAN\u0130 hastal\u0131\u011f\u0131 g\u00f6r\u00fcl\u00fcr. Bu hastal\u0131\u011f\u0131n belirtileri; kaslarda titremeler, kramplar, kaslarda a\u011fr\u0131l\u0131 kas\u0131lmalar, el ve ayak parmaklar\u0131nda i\u00e7e do\u011fru b\u00fck\u00fclmeler g\u00f6r\u00fcl\u00fcr.
\n Bir tetani krizi s\u0131ras\u0131nda elin durumu
\n Parathormonun Fazla Salg\u0131lanmas\u0131 Halinde;
\n Paratroid bezi b\u00fcy\u00fcr ve kanda Ca miktar\u0131 artar. \u0130skelet zay\u0131flar, kaslar, uyar\u0131lara \u00e7ok yava\u015f cavap verir. Kas zay\u0131flamas\u0131 (Atrofi) g\u00f6r\u00fcl\u00fcr. Kanda artan Ca b\u00f6breklere ta\u015f\u0131n\u0131r ve orada P iyonlar\u0131 ile birle\u015ferek b\u00f6brekta\u015flar\u0131n\u0131 olu\u015fturur. <\/p>\n","protected":false},"excerpt":{"rendered":"
A-) H\u0130POF\u0130Z: Beyinde, temel kemikteki T\u00fcrk e\u011feri denilen \u00e7ukur i\u00e7inde bulunur. \u0130nce bir sap ile hipotolamusa ba\u011fl\u0131d\u0131r. Di\u011fer i\u00e7 salg\u0131 bezlerinin \u00e7al\u0131\u015fmas\u0131 \u00fczerinde \u00f6nemli d\u00fczenleyici g\u00f6revi vard\u0131r. \u00d6n, ara ve arka lob olmak \u00fczere 3 k\u0131s\u0131mdan olu\u015fmu\u015ftur. Ara lob insanda sadece fet\u00fcste g\u00f6r\u00fcl\u00fcr. Ergin fertte iz halinde kal\u0131r. \u00d6nve arka loblar epitel, ara lob sinir …<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[1407,1403],"tags":[7897,9357,2510,9358,9192,9193,7976,9194,2636,9200],"class_list":["post-4200","post","type-post","status-publish","format-standard","hentry","category-fen-ve-teknoloji-odevleri","category-odevler","tag-arka-lob-hastaliklari","tag-asidofil-adenom","tag-beyin","tag-cushing-hastaligi","tag-endokrin-bez-hastaliklari","tag-hipotolamus","tag-jigantizm","tag-on-lob-hastaliklari","tag-somoatotropik","tag-tiroksin-hormonu"],"_links":{"self":[{"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/posts\/4200","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/comments?post=4200"}],"version-history":[{"count":0,"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/posts\/4200\/revisions"}],"wp:attachment":[{"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/media?parent=4200"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/categories?post=4200"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/www.islamidavet.com\/kutuphane\/wp-json\/wp\/v2\/tags?post=4200"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}